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At the same time, the absence of Gsr and TrxR in these cells results in an oxidized cytosol in which Grx1-disulfide or Grx-SSG accumulates

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Attenuation of Lipopolysaccharide-Induced Acute Lung Injury by Hispolon in Mice, Through Regulating the TLR4/PI3K/Akt/mTOR and Keap1/Nrf2/HO-1 Pathways, and Suppressing Oxidative Stress-Mediated ER Stress-Induced Apoptosis and Autophagy

Despite the lower incidence of NAFLD-related cirrhosis or hepatocellular carcinoma in comparison with other aetiologies, such as hepatitis, the exceptionally high prevalence and vast population at risk have propelled NAFLD to become the swiftest growing causative factor for hepatocellular carcinoma (Reference Huang, El-Serag and Loomba4)