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GSH depletion can trigger ferroptosis, which has been exploited in cancer treatments [17], and supplementation with GSH is utilized by glutathione peroxidase 4 (GPX4) to prevent lipid peroxidation, in turn inhibiting ferroptosis

The final step is deprotonation (Step 6, arrows K and L) to give the neutral primary amide

The increased skin growth and lack of cell death lead to the thickened areas of skin that are commonly associated with psoriasis

Initially characterized as a mitochondrially localized pro-apoptotic factor, in vitro and in vivo experiment reveal that myristoylation-mediated translocation to the plasma membrane converts FSP1 into a potent ferroptosis inhibitor (84, 85)