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N-acetylcysteine (NAC, a glutathione precursor) rescued IDH1/ cells under glucose withdrawal in these studies, and underscored the role that antioxidant defense plays in IDH1-driven adaptive survival

In vitro and animal data demonstrate enhanced cell survival, matrix production and gene activation when GHK-Cu is combined with light or antioxidant therapies

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This prevents neuronal ferroptosis induced by oxidative stress, alleviates glia overactivation, inhibits the secretion of pro-inflammatory factors, and regulates the inflammatory microenvironment