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10.1074/jbc.M308035200 37 FujiwaraS.KawazoeT.OhnishiK.KitagawaT.PopaC.VallsM.et al (2016)

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Savic-Radojevic, A

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This function may explain the ability of sorafenib to trigger caspase-independent cell death in certain cell types and enhance ROS accumulation in sorafenib-treated cancer patients [35, 36]

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It presents a lipid- and water-soluble substance meaning that it is active in both hydrophobic and hydrophilic environments (i.e

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