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Description

What makes it special is that it is designed to disrupt the interaction between FOXO4 and the tumor-suppressor protein p53 within senescent cells, thereby triggering programmed cell death (apoptosis) of those cells

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active ulcer, serious liver disease, hypotension, or gout)

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doi: 10.1002/ibd.20334

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Recent advances in vasoactive intestinal peptide physiology and pathophysiology: focus on the gastrointestinal system

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