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In luminal hypoxia, oxygen-deprived cells require sustenance to fulfill their elevated metabolic energy demands

This inactivation promotes C-X-C motif chemokine 5 (CXCL5), macrophage inflammatory protein-2 (MIPS2), keratinocytes-derived chemokine (KC), and tumor necrosis factor- (TNF-) activity, drawing leukocytes into the affected tissues [30]