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Given the availability of efficacious interventions for cigarette smoking cessation, concerted efforts should be made by healthcare providers and public health professionals to promote smoking cessation as a valuable approach for reducing non-smokers exposure to environmental tobacco smoke

The persistence of increased risk among former smokers is primarily due to the cumulative genetic and epigenetic damage caused by prolonged exposure to carcinogens in tobacco smoke

The harmful chemicals present in cigarette smoke impact fetal growth, disrupt placental function, and restrict oxygen supply to the developing baby

Further photographs, condition reports and in hand evaluations are available upon request