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Overall, the new data from the UK indicates that the harm caused by smoking appears to be cumulative

Abnormal M1/M2 macrophage phenotype profiles in the small airway wall and lumen in smokers and chronic obstructive pulmonary disease (COPD)

Given that PLWH are roughly twice as likely as the general population to smoke cigarettes [3,4,5] and have more trouble quitting [3, 4], and that nicotine (a primary constituent of tobacco smoke) is anti-inflammatory [6] and pro-cognitive [7], understanding the effects of being HIV+ and cigarette smoking on neuroinflammation and cognitive control in PLWH is crucial

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