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101 Whereas, the classical explanation is that, while hydroxychloroquine impaired phago/lysosomal function, it also appears to work in a lysosome-independent manner by impacting on intracellular TLRs, particularly TLR9, by inhibiting the production of TNF, and by interfering with the processing of the conversion of the membrane-bound pro-TNF into soluble mature protein

Reducing lactate production lowers H3K18la and suppresses tumor growth, while mechanistic work identified EP300-dependent H3K18la at the YTHDF2 promoter, increasing YTHDF2 and promoting decay of tumor-suppressive PER1 and TP53 transcripts, thereby facilitating malignancy [205]

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