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Activating mutations of phosphatidylinositol 4,5-bisphosphate 3-kinase catalytic subunit alpha (PIK3CA) gene confer sensitivity to PD-L1 checkpoint inhibition in metastatic adenocarcinoma patients

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Increased binding was present in the hippocampus, amygdala, thalamus, midbrain, pons, and cingulate cortex, although the exact area of the cingulate cortex was not defined

Its inhibition at the tissue level without systemic suppression represents a pharmacologically favorable profile for research applications where inflammation is driving local tissue damage