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By removing a hydrogen molecule from unsaturated lipids, these radicals produce a lipid radical (L ), which quickens the lipid peroxidation chain reaction [152]

This protective process supports multiple neurological functions, including: Reduced Neuroinflammation Calms inflammatory pathways that damage neurons

Mitochondria, ER, and nuclear membrane defects reveal early mechanisms for upper motor neuron vulnerability with respect to TDP-43 pathology

Anti-apoptotic protein upregulation Bcl-2 family regulation: Balance between pro-apoptotic (Bax, Bad, Bid) and anti-apoptotic (Bcl-2, Bcl-xL, Mcl-1) Bcl-2 family members determines cell survival versus death