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Pyridoxal-5-phosphate deficiency is associated with hyperhomocysteinemia regardless of antioxidant, thiamine, riboflavin, cobalamine, and folate status in critically ill patients

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P., Hogart, A

These pathological interwoven processes activate multimodal cell death pathways, such as apoptosis, necrosis, autophagy, and ferroptosis, posing a major challenge to neuroprotective treatment strategies