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Peptides as therapeutic agents: Challenges and opportunities in the green transition era

47,48 Dysregulated inflammatory signaling by fat tissue and release of growth factors, cytokines, and free radicals from the ovaries, liver, and other tissues contribute to the inflammatory state and promote further metabolic imbalance and insulin resistance

Effect of Tanshinone IIA for myocardial ischemia/reperfusion injury in animal model: preclinical evidence and possible mechanisms

Therapeutic targeting of mitochondrial metabolic dependencies CRC cells sustain a high metabolic state and therapeutic resistance by activating multiple mitochondrial pathways, including the TCA cycle, OXPHOS, FAO, and one-carbon metabolism [18]