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Inhibiting de novo lipogenesis identifies a therapeutic vulnerability in therapy-resistant colorectal cancer

They catalyze the thiolysis of 4-nitrophenyl acetate, exhibit thiol transferase activity, reduce trinitroglycerin, dehydroascorbic acid and monomethyl decanoic acid, and catalyze ethyl maleate and 5-3 isomerization of ketosteroids (8)

Identifying targetable metabolic dependencies across colorectal cancer progression

Secondly, there is an impaired synthesis of cystathionine from L-serine and homocysteine by cystathionine -synthase, leading to a subsequent reduction in the drainage of homocysteine from the methionine cycle to the transsulfuration pathway