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A number of pathologies have been characterized by an unbalanced cysteinome profile, including cystinuria, renal calculi, Huntington's disease, and Alzheimer's disease (68)

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The activation of these junctions may also induce long-term structural changes, such as the formation of tight associations with pericytes (which may encompass gene expression modulation), determining the stability and maturation of the vasculature [45], or short-lasting effects, like transient changes in endothelial permeability to solutes and circulating cells [46,47,48]

Schisterman EF, Sjaarda LA, Clemons T, Carrell DT, Perkins NJ, Johnstone E, et al