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The picture that emerges from these experiments is that endogenous GSH is not just an inhibitor of inflammation, but it is required to allow a proper response to infection, and direct the migration of inflammatory PMN away from the lung, where they cause ARDS, and towards the site of infection, where they kill bacteria (Figure 6)

In vitro studies of the effects of four complexing agents on the distribution and dialyzability of methylmercury in human blood

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Doses are adjusted by healthcare providers depending on medical condition, body weight, and treatment objectives