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Overexpression of miR-146a and miR-200b downregulates NR2A and NR2B proteins, contributing to cognitive deficits [71], while miR-137 controls excitatory synapse maturation and plasticity by regulating GluA1 expression, with its levels modulated by mGluR5 activation [63]

Among the genes evaluated, expression levels of genes were not significantly different in GHK and GHK-Cu group as compared to control

Aldose Reductase Inhibitors Given the relevance of aldose reductase action in the development of diabetic cataracts, the use of aldose reductase inhibitors has been suggested as a potential effective drug treatment against cataracts (Table 1)

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