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These results suggest a metabolic shift in ME/CFS patients from a hypometabolic state at baseline, characterized by down-regulated pathways to a hypermetabolic state post-exercise, characterized by over-activated pathways

In research models, this may include: Food intake behavior Meal-size signaling Satiety response after nutrient intake Delayed gastric emptying models Brain-based appetite regulation Hormonal feedback related to energy availability This makes cagrilintide useful for studying how peptide signals affect both biological function and behavioral outcomes related to food intake

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