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Postmenopausal osteoporosis (type I): After menopause, women experience a sudden drop in estrogen levels, a significant increase in osteoclast activity, accelerated bone resorption, insufficient osteoblast activity, and rapid loss of bone mass (30% -50% loss after 5-10 years of menopause), mainly in the lumbar spine and hip

Why it matters: Tissue repair is heavily dependent on highly specific receptor interactions

Animal and cell studies show shifts in enzymes that convert cholesterol to sex steroids, suggesting possible effects on estrogen and testosterone levels

KPV puts the literal brakes on this inflammatory response