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ROS, also as a second messenger, can activate tyrosine kinases and MAPK which promote cell development (81), and the protein kinase-B (Akt)/mTOR signaling pathway which promotes cell growth of gastric cancer (82)

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For example, the MEK inhibitor trametinib combined with 5-FU has been shown to synergistically suppress the MAPK pathway and enhance the response to neoadjuvant chemoradiotherapy [156]