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A cell with high NAD+ and low NADH is in an oxidized state, favoring catabolic pathways like fat oxidation and mitochondrial biogenesis

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To dissect the biochemical derangements imposed by chronic hyperglycaemia, a three-dimensional, tissue-engineered wound model has been engineered that faithfully recapitulates the accumulation of AGEs and the attendant impairment of fibroblast proliferation, angiogenic signaling, and extracellular-matrix turnover that underlie the stalled diabetic repair program (129)