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in this case, individuals show a limited or generalized deficiency of GSH and an accumulation of 5-oxoproline (in blood and cerebrospinal fluid) leading to metabolic acidosis [43], mental retardation, neuropsychiatric dysfunction, spinocerebellar degeneration, peripheral neuropathy, myopathy, hepato-splenomegaly, hemolytic anemia, aminoaciduria, and severe neurological complications [134]

To explain the similarities and differences in the phenotypic characteristics associated with DS, a gene dose imbalance theory has been hypothesized stating that patients with DS have an increased dose or number of gene copies on Hsa21, which may lead to increased gene expression

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The BPC 157 early regimen showed no changes at 30 min, and 5 h, and only mild congestion at 24 h of ligation time