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However, due to lower affinity for IGF-I, IGFBP-1 is usually in an unsaturated state and changing plasma concentrations of IGFBP-1 becomes important in determining the unbound fraction of IGF-I

infection dampens NLRP3 activation by modulating mitochondrial ROS production, partly through upregulation of antioxidant enzymes such as superoxide dismutases (SOD1 and SOD2), catalase (CAT), and glutathione peroxidase (GPx)