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TLR1/2/6 activation and subsequent signal transduction may be in part responsible for clinical immunopathological manifestations found in patients infected with COVID-19 (Gadanec et al., 2021)

doi: 10.3390/ijms20184472 Summary Keywords Glutathione, SARS-CoV-2, COVID-19, reactive oxygen species, oxidative stress, acute respiratory distress syndrome, atherosclerosis, atherothrombosis Citation Labarrere CA and Kassab GS (2022) Glutathione deficiency in the pathogenesis of SARS-CoV-2 infection and its effects upon the host immune response in severe COVID-19 disease