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Both GSH and OTC treatments caused a substantial increase in total glutathione (GSH and GSSG) contents 1 and 2 days after treatments, regardless whether or not the plants accumulate wild type levels of SA

Oxygen radicals and decreased GSH levels are implicated in the pathogenesis and progression of IPF [38]

Several metabolomics studies have confirmed that APAP-induced mitochondrial oxidative stress disrupted the metabolic processes of citric acid cycle and fatty acid -oxidation, thus blocking ATP synthesis and accelerating hepatocyte death

Radiation damage leads to a significant reduction in the abundance of bile acid-metabolizing commensal taxa (e.g., Lactobacillus and Bifidobacterium ) in the gut, thereby blocking the secondary bile acid production pathway and causing bile acid metabolism disorders 150