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It increases blood flow to injured areas, accelerating cellular repair and connective tissue regeneration all without the risks commonly associated with steroids or non-steroidal anti-inflammatory drugs (NSAIDs)

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29, 30, 31 The drug concentration ranges used were: BSO, 0400 M and L-PAM, 050 M (clinically achievable levels)

The transient elevation of peripheral cytokines, such as IL-6 induces sustained effects through the following mechanisms: 1) Activated microglia can sustain a pro-inflammatory phenotype over extended periods, thereby potentially becoming a continuous source of IL-1 and TNF- within the central nervous system (127, 128)