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In the context of AD, the inflammatory responses of M1 microglia, which are predominantly localized around the amyloid plaques, are initiated and sustained by P47 phagocyte oxidase and NF-B (nuclear factor kappa B) (130)

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postulated the role of indoxyl sulfate in the pathogenesis of renal anemia: indoxyl sulfate could stimulate oxidative stress and induce eryptosis through Organic Anion Transporter 2 (OAT2)- and NADPH oxidase-dependent, as well as GSH-independent mechanisms [257]