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Recent findings suggest that many of the memory enhancing effects initially attributed to AngII are likely due to the conversion of AngII to AngIV, and it is this peptide acting as an agonist at the AT 4 receptor subtype, that is responsible for cognitive facilitation (20, 57, 58)

Because inflammation is a core component and driver of NASH pathology, the anti-inflammatory actions of FGF21-based therapies are likely to benefit the general NASH patient population

Our data thus far show that 5FU drives increased mitochondrial biogenesis to address a functional requirement for increased mitochondrial ETC metabolism

EC/R group: Mice developed Ehrlich solid tumor were irradiated with a single dose of (0.5 Gy) gamma radiation as group 3