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The pathophysiology of diabetes involves a defective amplification of the late-phase insulin response to glucose by glucose-dependent insulinotropic polypeptide-regardless of etiology and phenotype

Furthermore, mitochondrial O-GlcNAc modification has been shown to enhance the therapeutic efficacy of transplanted mitochondria in a mouse model of stroke, while higher levels of O-GlcNAcylation in mitochondria correlating with improved clinical outcomes in SAH patients [177]

Pollard 2005 J Biomed Mater Res confirmed the acceleration of cutaneous healing in injured animals and humans

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