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151,152 H 2 S and cancer: beyond disturbed metabolism In addition to the energetic stimulus afforded by low H 2 S concentrations, the link between the adaptability of cancer cells to an evolving and challenging environment and enhanced H 2 S metabolism extends to adaptation to hypoxia, antioxidant capacity, neoangiogenesis, cell cycle regulation, apoptosis evasion and chemoresistance

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[11C]-Labeled metformin distribution in the liver and small intestine using dynamic positron emission tomography in mice demonstrates tissue-specific transporter dependency

Myeloid-derived suppressor cell membrane-coated magnetic nanoparticles for cancer theranostics by inducing macrophage polarization and synergizing immunogenic cell death