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Interestingly, TFR1 had been previously identified as the major cell surface receptor for IgA1 binding in renal mesangial cells and its overexpression has been correlated with the proliferation of mesangial cells in IgAN patients [269,270,271]

Oyama R

(73) believed that the important mechanism for NOR to exert anti-RA characteristics might be to prevent the release of IL-6 from FLSs, and its mechanism might be related to the inhibition of PKC/MAPKs/p65/cAMP response element-binding protein (CREB) pathway

If not closely managed, this can lead to side effects such as joint pain, fluid retention, insulin resistance, and other concerns