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These macrophage populations exhibit a pro-inflammatory M1-like phenotype and release the same inflammatory cytokines/chemokines characteristic of T1D development (24, 25)

This dysfunction creates a permissive environment for biofilm-forming pathogens such as S

In this article, we review efforts to develop new drugs targeting these processes, including agents that reactivate fetal haemoglobin, anti-sickling agents, anti-adhesion agents, modulators of ischaemiareperfusion and oxidative stress, agents that counteract free haemoglobin and haem, anti-inflammatory agents, anti-thrombotic agents and anti-platelet agents

Pharmacol Res (2019) 150:104497