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In animals exhibiting behavioral signs of tinnitus, an early, frequency-specific increase in the spontaneous firing rates of neurons in the DCN has been observed, 75 interpreted as a compensatory mechanism for reduced input from auditory nerve fibers originating in the cochlea

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They work by scavenging free radicals and reducing oxidative stress, thereby mitigating the risk of conditions such as heart disease, cancer, and neurodegenerative disorders