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Patricia Heitman, PharmD, is a Clinical Compounding Pharmacist at PCCA

Key messages Selenium (Se) and selenoproteins regulate ferroptosis, a lipid peroxidationdriven form of cell death

Antioxidant interventions that restore glutathione activity (including selenium supplementation, which activates GPx) have demonstrated significant reductions in TPO antibodies and oxidative stress markers in clinical trials

In vitro angiogenesis assays demonstrate that GHK-Cu (0.1-1 M) increases endothelial cell tubule formation by 60-90% compared to untreated controls, with effects mediated through VEGF-independent pathways involving integrin receptor activation and focal adhesion kinase signaling