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Our research confirms that cystine disrupts cellular redox levels by regulating GSH/GSSG, which in turn may enhance the Fenton reaction under antibiotic stress, thus creating favorable conditions for increased antibiotic-mediated bacterial death

With further in-depth analysis of its regulatory network and continued optimization of activator design, SIRT1 holds significant potential as a breakthrough target for multi-pathway, systemic, and precise intervention in MASLD

Abstract Background Recurrent pregnancy loss (RPL) presents one of the main problems in the field of reproductive medicine, due to the unknown etiology in 50% of cases, as well as limited evidence-based diagnostic and therapeutic modalities