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[76] demonstrated that AKT activated IKK which induced the non-canonical NF-B signaling by processing the NF-B2 (p100) protein to the p52 component of NF-B in HEK293 cells

The small increases at 60 and 90 days were likely analytical variation, not real chemical recovery

As expected, H 2 O 2 (0.2 mM) exposure disrupted cellular glutathione homeostasis in both HepG2 cells (a human hepatoma cell line) and NCTC1469 cells (a non-transformed mouse hepatocyte cell line), as demonstrated by an increase in the GSSG/GSH ratio over time (Fig