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40,41 In this review, we integrate recent advances from genetic, neuropathological, and neurobiochemical studies on ASD to further elucidate the pathogenetic mechanism at the molecular, cellular, and neural circuit levels

Furthermore, activation of the AMPK/ULK1 signaling pathway promotes selective ferroptosis of ferritin, leading to free iron release and lipid peroxidation accumulation, ultimately causing osteoblast ferroptosis (34)

This model simulates the human conditions of restricted blood flow and subsequent cognitive impairment due to carotid artery plaque build-up

It suppresses Erk1/2 and Akt phosphorylation generated by PDGF-BB