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Molecular pathways activated by low-dose carbon monoxide may slow the onset and limit the pathology in Parkinsons disease. These findings led Gomperts and his colleagues to test the effects of low doses of carbon monoxide in rodent models of Parkinsons

On average, they were 71 when the researchers looked at their surveys

Despite the obvious biologic plausibility, the lack of consistent epidemiological evidence might be due to methodological issues including lack of cohort follow-up time into older adulthood (when lung cancer usually becomes clinically manifest), small sample sizes, selection and recall biases, and lack of adjustment for tobacco use in the statistical modeling

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